Table 2

Effects of galactose elimination capacity, alcohol consumption, and plasma sodium concentration on the clinical course of alcoholic cirrhosis
Hepatic encephalopathy Ascites Variceal bleeding Death
All patients at risk Patients at risk + with GEC test All patients at risk Patients at risk + with GEC test All patients at risk Patients at risk + with GEC test All patients at risk Patients at risk + with GEC test
N = 417 N = 237 N = 164 N = 56 N = 410 N = 231 N = 466 N = 266
GEC, per 0.1 mmol/min loss 1.21 (1.11-1.31) 1.04 (0.84-1.30) 1.01 (0.95-1.08) 1.04 (0.98-1.10)
Alcohol consumption 0.93 (0.51-1.70) 0.82 (0.45-1.50) 1.95 (1.16-3.26) 3.18 (1.19-8.47) 2.01 (1.28-3.14) 2.78 (1.59-4.87) 2.20 (1.68-2.88) 2.45 (1.63-3.66)
Plasma sodium, per mmol/L loss 1.14 (1.10-1.19) 1.14 (1.09-1.18) 1.12 (1.05-1.20) 1.16 (1.06-1.28) 1.08 (1.05-1.11) 1.07 (1.04-1.10) 1.13 (1.11-1.15) 1.14 (1.11-1.18)

Effects are expressed as hazard ratios controlled for confounding by the two other patho-etiological measures, gender, age, and Charlson comorbidity index. Statistically significant associations are highlighted with bold font.

Jepsen et al.

Jepsen et al. BMC Research Notes 2012 5:509   doi:10.1186/1756-0500-5-509

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