Additional file 1: Table S1.
Demographic characteristics of subjects at inclusion. Table S2. Parent and product m/z values of LC-MS/MS analysis of eicosanoids. The eicosanoids in bold were selected for further processing. Table S3. Overview of selected genes related to inflammation and their primers; shaded genes are reference genes; genes in italic and with grey font did not pass quality control or were not considered for data analysis. Table S4. Gene-sets related to specialized biological functions and pathways selected from gene-array of a total of 85 genes. Table S5. Fold changes of genes. Figure S1. Statistical significance of repeated measurements, AUCmin, AUCplusand AUCtotal of inflammatory markers and clinical paramters (upper panel), oxylipins (middle panel) and gene expression data (lower panel) after OGTT, OLTT and OG + LTT versus control, green p < 0.05, yellow p < 0.01, orange p < 0.001. Figure S2. Mean ± SEM postprandial relative EPA, DHA, 17(S)-HDoHE, 17-keto-DHA in healthy subjects (N = 12) after ingestion of water (□), OGTT (◊), OLTT (△), OG + LTT (○). ANOVA: OGTT, OLTT, OL + GTT versus water: green p < 0.05, yellow p < 0.01, orange p < 0.001; water versus baseline; * p < 0.05, ** p < 0.01, *** p < 0.001.
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Wopereis et al. BMC Medical Genomics 2013 6:44 doi:10.1186/1755-8794-6-44