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Exon expression in lymphoblastoid cell lines from subjects with schizophrenia before and after glucose deprivation

Maureen V Martin1, Brandi Rollins1, P Adolfo Sequeira1, Andrea Mesén23, William Byerley4, Richard Stein1, Emily A Moon1, Huda Akil5, Edward G Jones6, Stanley J Watson5, Jack Barchas7, Lynn E DeLisi8, Richard M Myers9, Alan Schatzberg10, William E Bunney1 and Marquis P Vawter1*

Author Affiliations

1 Department of Psychiatry and Human Behavior, Univ. of California, Irvine, CA, USA

2 Psychiatric Genetics Research Center, Heredia, Costa Rica

3 Hospital Nacional Psiquiatrico, Pavas, San Jose, Costa Rica

4 Psychiatry, Univ. of California, San Francisco, CA, USA

5 Molecular & Behavioral Neuroscience Institute, Univ. of Michigan, MI, USA

6 Neuroscience Center, Univ. of California Davis, CA, USA

7 Psychiatry, Cornell Univ., New York NY, USA

8 Psychiatry, New York Univ., New York NY, USA

9 Hudson Alpha, Huntsville AB, USA

10 Psychiatry, Stanford University Palo Alto, CA, USA

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BMC Medical Genomics 2009, 2:62  doi:10.1186/1755-8794-2-62

Published: 22 September 2009

Additional files

Additional File 1:

Primer sequences. The tables lists primer sequences for all primers used in this study.

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Additional File 2:

significant diagnosis, glucose deprivation and probeset interaction effects on transcript expression overlap with previous findings in the prefrontal cortex of subjects with schizophrenia compared to controls. Tables list transcripts with significant diagnosis × probeset, glucose deprivation × probeset and diagnosis × glucose deprivation × probeset effects, and overlap with previous findings in the prefrontal cortex of subjects with schizophrenia compared to controls.

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Additional File 3:

PPARGC1B and FBXW5 expression. Figures displaying significant Diagnosis × Glucose deprivation × Probeset interaction effects on transcripts expression for genes PPARGC1B and FBXW5.

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