Table 2

ACE genotype and allele distributions among controls and migraine patients in a Norwegian population



Genotypes
Alleles


N
DD(%)
ID(%)
II(%)
D(%)
I(%)

Controls
403
92 (26.6)
204 (50.6)
107 (22.8)
388 (48.1)
418 (51.9)
Migraine
347
78 (22.5)
186 (53.6)
83 (23.9)
342 (49.3)
352 (50.7)
MwA subgroup
155
34 (21.9)
87 (56.1)
34 (21.9)
155 (50.0)
155 (50.0)
MoA subgroup
187
43 (23.0)
96 (51.3)
48 (25.7)
182 (48.7)
192 (51.3)
Lisinopril responders
12
2 (16.7)
6 (50.0)
4 (33.3)
10 (41.7)
14 (58.3)
Lisinopril non-responders
37
10 (27.0)
16 (43.2)
11 (29.7)
36 (48.6)
38 (51.4)
Candesartan responders*
18
7 (38.9)
9 (50.0)
2 (11.1)
23 (63.9)
13 (36.1)
Candesartan non-responders*
38
8 (21.1)
18 (47.4)
12 (31.6)
34 (44.7)
42 (55.3)
Responders combined
30
9 (30.0)
15 (50.0)
6 (20.0)
33 (55.0)
27 (45.0)
Non-responders combined
75
18 (24.0)
34 (45.3)
23 (30.7)
70 (46.7)
80 (53.3)

* Response data available in 56 of 59 genotyped

Allele and genotype frequency distributions are not significantly different for any diagnostic groups (migraine, MwA, MoA) vs controls, or for responders vs non-responders (p > 0.05).

Tronvik et al. BMC Neurology 2008 8:4   doi:10.1186/1471-2377-8-4