Additional file 2: Figure S1.
IP3R3 -positive microvillous cells remained in the Skn-1a-/- MOE. Effects of Skn-1a deficiency on IP3R3 -positive microvillous cells were examined by immunostaining with an anti-Trpm5 (green) and an anti-IP3R3 (red) antibodies using coronal sections of the wild-type and Skn-1a-/- MOE of adult mice. Trpm5-positive cells did not overlap with IP3R3-positive cells in the wild-type MOE. In the Skn-1a-/- MOE, IP3R3-positive cells were observed, whereas no immunoreactive signal for Trpm5 was detected, suggesting that one of remaining superficial microvillous cells in the Skn-1a-/- MOE would be IP3R3-microvillous cells, and Skn-1a is not essential to generate them. Scale bars, 10 μm.
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Yamaguchi et al. BMC Neuroscience 2014 15:13 doi:10.1186/1471-2202-15-13