Research article
Marek’s disease virus infection induces widespread differential chromatin marks in inbred chicken lines
- † Equal contributors
1 Department of Animal & Avian Sciences, University of Maryland, College Park, MD, USA
2 USDA, ARS, Avian Disease and Oncology Laboratory, East Lansing, MI, USA
3 Laboratory of Molecular Immunology, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, MD, USA
BMC Genomics 2012, 13:557 doi:10.1186/1471-2164-13-557
Published: 16 October 2012Additional files
Additional file 1:
Figures S1 to S9. Supplementary Figures S1 to S9.
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Additional file 2:
Differential H3K4me3 marks. Genome-wide differential H3K4me3 marks produced by DESeq (FDR < 0.4) and the associated genes. P-values from three contrasts are displayed as follows: 63: 63I vs 63N, 72: 72I vs 72N, 63v72N: 63N vs 72N. 63I: line 63 infected, 63N: line 63 control, 72I: line 72 infected, 72N: line 72 control.
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Additional file 3:
Differential H3K27me3 marks.Genome-wide differential H3K27me3 marks produced by DESeq (FDR < 0.4) and the associated genes. P-values from three contrasts are displayed as follows: 63: 63I vs 63N, 72: 72I vs 72N, 63v72N: 63N vs 72N. 63I: line 63 infected, 63N: line 63 control, 72I: line 72 infected, 72N: line 72 control.
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Additional file 4:
Putative bivalent genes from colocalization analysis of H3K4me3 and H3K27me3.
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Additional file 5:
Functional annotation clustering of bivalent genes using DAVID.
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Additional file 6:
Sequencing results showing raw and mapped reads for each sample.
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Additional file 7:
Primers used for quantitative PCR validation.
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