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Genetic interactions between ntf-2, chic, and Sd. The genetic crosses performed (top of the table). #s in parenthesis indicate columns in the table. The progeny resulting from the cross of ntf-2/FM7 females to males carrying the suppressing chromosome were counted. Four alleles of ntf-2 are listed in column 1. The genotype of the suppressing chromosomes are presented in the column 2. The numbers of trans-heterozygous females (ntf-2/+; Su/+; column 3), of non-mutant males (FM7/Y; Su/+, column 4), and of potentially suppressed mutant males (ntf-2/Y; Su/+) are presented. The ntf-2/+; Su/+ males were divided into two columns (5 and 6) depending on their eye phenotype and the viability of these males is indicated (100% X ntf-2 males/ntf-2/+ females). The number of progeny of the cross shown on the top and marked by * are not shown. |
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| Parents |
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| ntf-2 (1)/FM7c; +/+ |
× |
+/Y; Su(2)/Balancer |
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| Progeny |
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| ntf-2/+; Su/+(3) |
ntf-2/Y; Su/+ (5, 6) |
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| FM7c/+; Su/+ * |
FM7c/Y; Su/+ (4) |
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| ntf-2/+; Balancer/+* |
ntf-2/Y; Balancer/+* |
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| FM7c/+; Balancer/+* |
FM7c/Y; Balancer/+ |
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|
|
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| ntf-2 allele (1) |
Su (2) chromosome Cytology (2) |
ntf-2/ +; Su/+ (3) |
FM7/Y;Su/+ (4) |
ntf-2/ Y; Su/+ small eyes (5) |
ntf-2/Y; Su/+ normal eyes (6) |
ntf-2/Y; viability |
Suppression of ntf-2 eye |
|
|
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| P49 |
+ or Balancer |
508 |
328 |
72 |
0 |
14% |
- |
| P7 |
+ or Balancer |
521 |
325 |
62 |
0 |
12% |
- |
| G0086 |
+ or Balancer |
503 |
154 |
42 |
0 |
8% |
- |
| G0337 |
+ or Balancer |
452 |
180 |
39 |
0 |
9% |
- |
| P49 |
Df(2R)Px2 60B; 60D |
50 |
13 |
4 |
19 |
46% |
+ |
| P7 |
Df(2R)Px2 60B; 60D |
104 |
28 |
10 |
1 |
11% |
+ |
| P49 |
In(2LR)Px[4] 60C-60D,21-22A |
51 |
14 |
4 |
15 |
39% |
+ |
| P7 |
In(2LR)Px[4] 60C-60D,21-22A |
155 |
53 |
6 |
1 |
5% |
+ |
| P7 |
P{lacW}l(2)04111 [k13009] 22A |
68 |
24 |
0 |
4 |
6% |
+ |
| P49 |
Df(2L)cl-h2 25D; 25F |
9 |
5 |
0 |
10 |
100% |
+ |
| P7 |
Df(2L)cl-h2 25D; 25F |
23 |
13 |
0 |
13 |
56% |
+ |
| P49 |
Df(2L)GpdhA 25D; 26A |
66 |
14 |
2 |
2 |
6% |
+ |
| P7 |
Df(2L)GpdhA 25D; 26A |
49 |
27 |
1 |
2 |
6% |
+ |
| P49 |
chicK13321 26A |
53 |
25 |
3 |
1 |
8% |
+ |
| P7 |
chicK13321 26A |
67 |
40 |
1 |
11 |
18% |
+ |
| P49 |
chic221 26A |
30 |
12 |
0 |
10 |
33% |
+ |
| P7 |
chic221 26A |
65 |
48 |
8 |
3 |
17% |
+ |
| G0086 |
chic221 26A |
43 |
22 |
0 |
6 |
14% |
+ |
| G0337 |
chic221 26A |
69 |
44 |
2 |
4 |
9% |
+ |
| P49 |
chic01320 26A |
46 |
35 |
0 |
4 |
9% |
+ |
| P49 |
chic2 26A |
74 |
68 |
0 |
3 |
4% |
+ |
| P7 |
chic2 26A |
43 |
33 |
4 |
1 |
12% |
+ |
| P49 |
In(2R)SD72, In(2R)NS, Sd[72] |
35 |
30 |
0 |
12 |
34% |
+ |
| G0086 |
In(2R)SD72, In(2R)NS, Sd[72] |
40 |
7 |
0 |
2 |
5% |
+ |
| P49 |
UAS-RanGAP12A-6, hsp70-GAL4 |
16 |
6 |
0 |
5 |
31% |
+ |
| P7 |
UAS-RanGAP12A-6, hsp70-GAL4 |
26 |
11 |
0 |
7 |
27% |
+ |
| P7 |
UAS-RanGAP12A-6, arm-GAL4 |
28 |
12 |
1 |
4 |
18% |
+ |
Minakhina et al. BMC Cell Biology 2005 6:32 doi:10.1186/1471-2121-6-32 |
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