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8 result(s) for 'author#Janos Groh' within BMC

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  1. From october 25th - 27th 2013, the 5th NEUROWIND e.V. meeting was held in Motzen, Brandenburg, Germany. This year more than 60 doctoral students and postdocs from over 25 different groups working in German uni...

    Authors: Ralf A Linker, Tim Magnus, Thomas Korn, Christoph Kleinschnitz and Sven G Meuth
    Citation: Experimental & Translational Stroke Medicine 2013 5:15
  2. The neuronal ceroid lipofuscinoses constitute a group of fatal inherited lysosomal storage diseases that manifest in profound neurodegeneration in the CNS. Visual impairment usually is an early symptom and sel...

    Authors: Janos Groh, David Stadler, Mathias Buttmann and Rudolf Martini
    Citation: Acta Neuropathologica Communications 2014 2:54
  3. The neuronal ceroid lipofuscinoses (CLN diseases) are fatal lysosomal storage diseases causing neurodegeneration in the CNS. We have previously shown that neuroinflammation comprising innate and adaptive immun...

    Authors: Kristina Berve, Brian L. West, Rudolf Martini and Janos Groh
    Citation: Journal of Neuroinflammation 2020 17:323
  4. Promotion of myelin repair in the context of demyelinating diseases such as multiple sclerosis (MS) still represents a clinical unmet need, given that this disease is not only characterized by autoimmune activ...

    Authors: Peter Göttle, Janos Groh, Laura Reiche, Joel Gruchot, Nicole Rychlik, Luisa Werner, Iria Samper Agrelo, Rainer Akkermann, Annika Zink, Alessandro Prigione, Hans-Peter Hartung, Rudolf Martini and Patrick Küry
    Citation: Journal of Neuroinflammation 2023 20:7
  5. We could previously identify components of both the innate and the adaptive immune system as disease modifiers in the pathogenesis of models for Charcot-Marie-Tooth (CMT) neuropathies type 1B and 1X. As part o...

    Authors: Dennis Klein, Janos Groh, Andreas Weishaupt and Rudolf Martini
    Citation: Journal of Neuroinflammation 2015 12:49
  6. Isolated generalized dystonia is a central motor network disorder characterized by twisted movements or postures. The most frequent genetic cause is a GAG deletion in the Tor1a (DYT1) gene encoding torsinA with a...

    Authors: Chi Wang Ip, Ioannis U. Isaias, Burak B. Kusche-Tekin, Dennis Klein, Janos Groh, Aet O’Leary, Susanne Knorr, Takahiro Higuchi, James B. Koprich, Jonathan M. Brotchie, Klaus V. Toyka, Andreas Reif and Jens Volkmann
    Citation: Acta Neuropathologica Communications 2016 4:108