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Genomic patterns of pathogen evolution revealed by comparison of Burkholderia pseudomallei, the causative agent of melioidosis, to avirulent Burkholderia thailandensis.
Yu Y, Kim HS, Chua HH, Lin CH, Sim SH, Lin D, Derr A, Engels R, DeShazer D, Birren B, Nierman WC, Tan P
BMC Microbiol
2006,
6
:46
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PubMed Central articles that cite the above article:
1.
A horizontal gene transfer event defines two distinct groups within Burkholderia pseudomallei that have dissimilar geographic distributions.
Tuanyok A, Auerbach RK, Brettin TS, Bruce DC, Munk AC, Detter JC, Pearson T, Hornstra H, Sermswan RW, Wuthiekanun V, Peacock SJ, Currie BJ, Keim P, Wagner DM
J Bacteriol
2007 Dec,
189
:9044-9
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2.
Genetic tools for select-agent-compliant manipulation of Burkholderia pseudomallei.
Choi KH, Mima T, Casart Y, Rholl D, Kumar A, Beacham IR, Schweizer HP
Appl Environ Microbiol
2008 Feb,
74
:1064-75
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3.
Targeted mutagenesis of Burkholderia thailandensis and Burkholderia pseudomallei through natural transformation of PCR fragments.
Thongdee M, Gallagher LA, Schell M, Dharakul T, Songsivilai S, Manoil C
Appl Environ Microbiol
2008 May,
74
:2985-9
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4.
The core and accessory genomes of Burkholderia pseudomallei: implications for human melioidosis.
Sim SH, Yu Y, Lin CH, Karuturi RK, Wuthiekanun V, Tuanyok A, Chua HH, Ong C, Paramalingam SS, Tan G, Tang L, Lau G, Ooi EE, Woods D, Feil E, Peacock SJ, Tan P
PLoS Pathog
2008 Oct,
4
:e1000178
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