Log on / register
Feedback | Support | My details
Molecular breakpoint cloning and gene expression studies of a novel translocation t(4;15)(q27;q11.2) associated with Prader-Willi syndrome.
Schüle B, Albalwi M, Northrop E, Francis DI, Rowell M, Slater HR, Gardner RJ, Francke U
BMC Med Genet 2005 May 6, 6:18
[Full text] [PubMed] [Related articles]


printer friendly 
to  [Help]

PubMed Central articles that cite the above article:

1.
SnoRNA Snord116 (Pwcr1/MBII-85) deletion causes growth deficiency and hyperphagia in mice.
Ding F, Li HH, Zhang S, Solomon NM, Camper SA, Cohen P, Francke U
PLoS One 2008, 3:e1709
 [PubMed Central] [PubMed] [Related articles]
2.
Deletion of the MBII-85 snoRNA gene cluster in mice results in postnatal growth retardation.
Skryabin BV, Gubar LV, Seeger B, Pfeiffer J, Handel S, Robeck T, Karpova E, Rozhdestvensky TS, Brosius J
PLoS Genet 2007 Dec 28, 3:e235
 [PubMed Central] [PubMed] [Related articles]
3.
Prader-Willi phenotype caused by paternal deficiency for the HBII-85 C/D box small nucleolar RNA cluster.
Sahoo T, del Gaudio D, German JR, Shinawi M, Peters SU, Person RE, Garnica A, Cheung SW, Beaudet AL
Nat Genet 2008 Jun, 40:719-21
 [PubMed Central] [PubMed] [Related articles]
printer friendly 
to  [Help]
Terms and Conditions Privacy statement Information for advertisers Jobs at BMC Contact us
© 1999-2010 BioMed Central Ltd unless otherwise stated. Part of Springer Science+Business Media.