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Production of infectious human immunodeficiency virus type 1 does not require depletion of APOBEC3G from virus-producing cells.
Kao S, Miyagi E, Khan MA, Takeuchi H, Opi S, Goila-Gaur R, Strebel K
Retrovirology
2004,
1
:27
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PubMed Central articles that cite the above article:
1.
Population level analysis of human immunodeficiency virus type 1 hypermutation and its relationship with APOBEC3G and vif genetic variation.
Pace C, Keller J, Nolan D, James I, Gaudieri S, Moore C, Mallal S
J Virol
2006 Sep,
80
:9259-69
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Polymorphisms of CUL5 are associated with CD4+ T cell loss in HIV-1 infected individuals.
An P, Duggal P, Wang LH, O'Brien SJ, Donfield S, Goedert JJ, Phair J, Buchbinder S, Kirk GD, Winkler CA
PLoS Genet
2007 Jan 26,
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Newly synthesized APOBEC3G is incorporated into HIV virions, inhibited by HIV RNA, and subsequently activated by RNase H.
Soros VB, Yonemoto W, Greene WC
PLoS Pathog
2007 Feb,
3
:e15
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*** Unable to retrieve article 17291161 ***
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Human immunodeficiency virus type 1 Vif inhibits packaging and antiviral activity of a degradation-resistant APOBEC3G variant.
Opi S, Kao S, Goila-Gaur R, Khan MA, Miyagi E, Takeuchi H, Strebel K
J Virol
2007 Aug,
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Vif counteracts a cyclophilin A-imposed inhibition of simian immunodeficiency viruses in human cells.
Takeuchi H, Buckler-White A, Goila-Gaur R, Miyagi E, Khan MA, Opi S, Kao S, Sokolskaja E, Pertel T, Luban J, Strebel K
J Virol
2007 Aug,
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T cells contain an RNase-insensitive inhibitor of APOBEC3G deaminase activity.
Thielen BK, Klein KC, Walker LW, Rieck M, Buckner JH, Tomblingson GW, Lingappa JR
PLoS Pathog
2007 Sep 21,
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8.
Production of infectious virus and degradation of APOBEC3G are separable functional properties of human immunodeficiency virus type 1 Vif.
Kao S, Goila-Gaur R, Miyagi E, Khan MA, Opi S, Takeuchi H, Strebel K
Virology
2007 Dec 20,
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:329-39
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9.
Enzymatically active APOBEC3G is required for efficient inhibition of human immunodeficiency virus type 1.
Miyagi E, Opi S, Takeuchi H, Khan M, Goila-Gaur R, Kao S, Strebel K
J Virol
2007 Dec,
81
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10.
Identification of an APOBEC3G binding site in human immunodeficiency virus type 1 Vif and inhibitors of Vif-APOBEC3G binding.
Mehle A, Wilson H, Zhang C, Brazier AJ, McPike M, Pery E, Gabuzda D
J Virol
2007 Dec,
81
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12.
Vpr14-88-Apobec3G fusion protein is efficiently incorporated into Vif-positive HIV-1 particles and inhibits viral infection.
Ao Z, Yu Z, Wang L, Zheng Y, Yao X
PLoS One
2008,
3
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13.
HIV-1 Vif promotes the formation of high molecular mass APOBEC3G complexes.
Goila-Gaur R, Khan MA, Miyagi E, Kao S, Opi S, Takeuchi H, Strebel K
Virology
2008 Mar 1,
372
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14.
APOBEC3G is degraded by the proteasomal pathway in a Vif-dependent manner without being polyubiquitylated.
Dang Y, Siew LM, Zheng YH
J Biol Chem
2008 May 9,
283
:13124-31
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16.
APOBEC3G and APOBEC3F require an endogenous cofactor to block HIV-1 replication.
Han Y, Wang X, Dang Y, Zheng YH
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2008 Jul,
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17.
Differential sensitivity of "old" versus "new" APOBEC3G to human immunodeficiency virus type 1 vif.
Goila-Gaur R, Khan MA, Miyagi E, Strebel K
J Virol
2009 Jan,
83
:1156-60
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18.
Regulation of APOBEC3 proteins by a novel YXXL motif in human immunodeficiency virus type 1 Vif and simian immunodeficiency virus SIVagm Vif.
Pery E, Rajendran KS, Brazier AJ, Gabuzda D
J Virol
2009 Mar,
83
:2374-81
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