Log on / register
Feedback | Support | My details
Immunopathogenesis of brain abscess.
Kielian T
J Neuroinflammation 2004 Aug 17, 1:16
[Full text] [PubMed] [Related articles]


printer friendly 
to  [Help]

PubMed Central articles that cite the above article:

1.
Induction of proinflammatory mediators requires activation of the TRAF, NIK, IKK and NF-kappaB signal transduction pathway in astrocytes infected with Escherichia coli.
Kim JM, Oh YK, Lee JH, Im DY, Kim YJ, Youn J, Lee CH, Son H, Lee YS, Park JY, Choi IH
Clin Exp Immunol 2005 Jun, 140:450-60
 [PubMed Central] [PubMed] [Related articles]
2.
Minocycline modulates neuroinflammation independently of its antimicrobial activity in staphylococcus aureus-induced brain abscess.
Kielian T, Esen N, Liu S, Phulwani NK, Syed MM, Phillips N, Nishina K, Cheung AL, Schwartzman JD, Ruhe JJ
Am J Pathol 2007 Oct, 171:1199-214
 [PubMed Central] [PubMed] [Related articles]
3.
MyD88-dependent signals are essential for the host immune response in experimental brain abscess.
Kielian T, Phulwani NK, Esen N, Syed MM, Haney AC, McCastlain K, Johnson J
J Immunol 2007 Apr 1, 178:4528-37
 [PubMed Central] [PubMed] [Related articles]
4.
Recognition of Staphylococcus aureus-derived peptidoglycan (PGN) but not intact bacteria is mediated by CD14 in microglia.
Esen N, Kielian T
J Neuroimmunol 2005 Dec 30, 170:93-104
 [PubMed Central] [PubMed] [Related articles]
5.
The synthetic peroxisome proliferator-activated receptor-gamma agonist ciglitazone attenuates neuroinflammation and accelerates encapsulation in bacterial brain abscesses.
Kielian T, Syed MM, Liu S, Phulwani NK, Phillips N, Wagoner G, Drew PD, Esen N
J Immunol 2008 Apr 1, 180:5004-16
 [PubMed Central] [PubMed] [Related articles]
6.
Staphylococcus aureus-derived peptidoglycan induces Cx43 expression and functional gap junction intercellular communication in microglia.
Garg S, Md Syed M, Kielian T
J Neurochem 2005 Oct, 95:475-83
 [PubMed Central] [PubMed] [Related articles]
7.
The role of Toll-like receptors in CNS response to microbial challenge.
Konat GW, Kielian T, Marriott I
J Neurochem 2006 Oct, 99:1-12
 [PubMed Central] [PubMed] [Related articles]
8.
15-deoxy-Delta12,14-prostaglandin J2 (15d-PGJ2) and ciglitazone modulate Staphylococcus aureus-dependent astrocyte activation primarily through a PPAR-gamma-independent pathway.
Phulwani NK, Feinstein DL, Gavrilyuk V, Akar C, Kielian T
J Neurochem 2006 Dec, 99:1389-1402
 [PubMed Central] [PubMed] [Related articles]
9.
Central role for MyD88 in the responses of microglia to pathogen-associated molecular patterns.
Esen N, Kielian T
J Immunol 2006 Jun 1, 176:6802-11
 [PubMed Central] [PubMed] [Related articles]
10.
Serial magnetic resonance imagings of multiple brain abscesses in a patient with pneumococcal meningoencephalitis.
Hong JM, Choi JY, Park HY, Huh K
J Korean Med Sci 2008 Dec, 23:1102-4
 [PubMed Central] [PubMed] [Related articles]
11.
Glial connexins and gap junctions in CNS inflammation and disease.
Kielian T
J Neurochem 2008 Aug, 106:1000-16
 [PubMed Central] [PubMed] [Related articles]
12.
Poly (ADP-ribose) polymerases (PARPs) 1-3 regulate astrocyte activation.
Phulwani NK, Kielian T
J Neurochem 2008 Jul, 106:578-90
 [PubMed Central] [PubMed] [Related articles]
13.
TLR2 expression in astrocytes is induced by TNF-alpha- and NF-kappa B-dependent pathways.
Phulwani NK, Esen N, Syed MM, Kielian T
J Immunol 2008 Sep 15, 181:3841-9
 [PubMed Central] [PubMed] [Related articles]
14.
MyD88 expression by CNS-resident cells is pivotal for eliciting protective immunity in brain abscesses.
Garg S, Nichols JR, Esen N, Liu S, Phulwani NK, Syed MM, Wood WH, Zhang Y, Becker KG, Aldrich A, Kielian T
ASN Neuro 2009, 1:
 [PubMed Central] [PubMed] [Related articles]
15.
A protective role for ELR+ chemokines during acute viral encephalomyelitis.
Hosking MP, Liu L, Ransohoff RM, Lane TE
PLoS Pathog 2009 Nov, 5:e1000648
 [PubMed Central] [PubMed] [Related articles]
printer friendly 
to  [Help]
Terms and Conditions Privacy statement Information for advertisers Jobs at BMC Contact us
© 1999-2009 BioMed Central Ltd unless otherwise stated. Part of Springer Science+Business Media.