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Production of infectious human immunodeficiency virus type 1 does not require depletion of APOBEC3G from virus-producing cells.
Kao S, Miyagi E, Khan MA, Takeuchi H, Opi S, Goila-Gaur R, Strebel K
Retrovirology 2004, 1:27
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BioMed Central articles that cite the above article:

1.


Research    
A novel HIV-1 restriction factor that is biologically distinct from APOBEC3 cytidine deaminases in a human T cell line CEM.NKR
Zhou T, Han Y, Dang Y, Wang X, Zheng YH
Retrovirology 2009, 6:31 (3 April 2009)
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2.


Research    
APOBEC3G induces a hypermutation gradient: purifying selection at multiple steps during HIV-1 replication results in levels of G-to-A mutations that are high in DNA, intermediate in cellular viral RNA, and low in virion RNA
Russell RA, Moore MD, Hu WS, Pathak VK
Retrovirology 2009, 6:16 (13 February 2009)
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3.


Review    
HIV-1 Vif, APOBEC, and Intrinsic Immunity
Goila-Gaur R, Strebel K
Retrovirology 2008, 5:51 (24 June 2008)
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4.


Research    
The dimerization domain of HIV-1 viral infectivity factor Vif is required to block virion incorporation of APOBEC3G
Miller JH, Presnyak V, Smith HC
Retrovirology 2007, 4:81 (24 November 2007)
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5.


Research    
Targeting APOBEC3A to the viral nucleoprotein complex confers antiviral activity
Goila-Gaur R, Khan MA, Miyagi E, Kao S, Strebel K
Retrovirology 2007, 4:61 (29 August 2007)
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6.


Review    
Intracellular immunity to HIV-1: newly defined retroviral battles inside infected cells
Zheng YH, Peterlin BM
Retrovirology 2005, 2:25 (13 April 2005)
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7.


Commentary    
HIV-1 Vif and APOBEC3G: Multiple roads to one goal
Goncalves J, Santa-Marta M
Retrovirology 2004, 1:28 (21 September 2004)
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This article is also cited by a further 18 articles in PubMed Central

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