University of Cincinnati, United States of America
Molecular Pain considers manuscripts in pain research at the cellular, subcellular and molecular levels. Molecular Pain provides a forum for molecular pain scientists to communicate their research findings in a targeted manner to others in this important and growing field.
In our journey to explore molecular pain mechanisms and to identify effective targets for pain management, it is important for scientists around the world to have a rapid and freely accessible forum for exchanging ideas, debating hot topics, developing collaborations, promoting science, and improving pain medicine. As an online, Open Access journal, Molecular Pain will help to fulfill these goals.
Dr Jianguo Gu is currently a Professor of Anaesthesiology, in the Department of Anaesthesiology, College of Medicine at the University of Cincinnati in Ohio. He is also the founder of Molecular Pain.
Dr. Gu was born in Nanjing, a capital city of Jiangsu Province in China. He was graduated from Shanghai First Medical College in 1985. He spent the next 4 years at China Pharmaceutical University, where he studied clinical pharmacology. In 1989, he went to Canada and entered a graduate program at the University of Manitoba to study neuropharmacology in Dr. Jonathan Geiger’s laboratory, where he received his Ph.D. degree in 1994. He then spent 4 years as a research associate in Dr. Amy MacDermott’s laboratory to study neuronal and molecular mechanisms of pain at the College of Physicians & Surgeons of Columbia University in New York. In 1998, he took a faculty position at the University of Florida where he established a research laboratory in the McKnight Brain Institute and Department of Oral Surgery. He dedicated himself to research, teaching and service for 11 years at the University of Florida before moving to the University of Cincinnati on August 1, 2009.
Dr. Gu’s main research interests are cellular and molecular mechanisms of inflammatory and neuropathic pain. His research has focused on a number of receptor families such as glutamate receptors, purinergic receptors, and TRP channels and their roles in sensory signaling and pain.