BMC Cancer

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Novel markers for differentiation of lobular and ductal invasive breast carcinomas by laser microdissection and microarray analysis

Gulisa Turashvili, Jan Bouchal*, Karl Baumforth, Wenbin Wei, Marta Dziechciarkova, Jiri Ehrmann, Jiri Klein, Eduard Fridman, Jozef Skarda, Josef Srovnal, Marian Hajduch, Paul Murray and Zdenek Kolar

BMC Cancer 2007, 7:55 doi:10.1186/1471-2407-7-55

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Research article   Open Access

Association of HADHA expression with the risk of breast cancer: targeted subset analysis and meta-analysis of microarray data

Manju Mamtani, Hemant Kulkarni BMC Research Notes 2012, 5:25 (12 January 2012)

Research   Open Access

Bimodal gene expression patterns in breast cancer

Marina Bessarabova, Eugene Kirillov, Weiwei Shi, Andrej Bugrim, Yuri Nikolsky, Tatiana Nikolskaya BMC Genomics 2010, 11(Suppl 1):S8 (10 February 2010)

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Increased levels of active c-Src distinguish invasive from in situ lobular lesions

Donghui Zou, Han-Seung Yoon, Ahmad Anjomshoaa, David Perez, Ryuji Fukuzawa, Parry Guilford, Bostjan Humar Breast Cancer Research 2009, 11:R45 (7 July 2009)

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Protein kinase D1 regulates matrix metalloproteinase expression and inhibits breast cancer cell invasion

Tim Eiseler, Heike Döppler, Irene K Yan, Steve Goodison, Peter Storz Breast Cancer Research 2009, 11:R13 (25 February 2009)

Protein kinase D1 is identified as a marker for invasive breast cancer, while loss of PKD1 expression may increase the malignant potential of breast cancer cells, suggesting re-expression of PKD1 as a possible therapeutic strategy.

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A resampling-based meta-analysis for detection of differential gene expression in breast cancer

Bala Gur-Dedeoglu, Ozlen Konu, Serkan Kir, Ahmet Ozturk, Betul Bozkurt, Gulusan Ergul, Isik G Yulug BMC Cancer 2008, 8:396 (30 December 2008)

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A functional Notchsurvivin gene signature in basal breast cancer

Connie W Lee, Karl Simin, Qin Liu, Janet Plescia, Minakshi Guha, Ashraf Khan, Chung-Cheng Hsieh, Dario C Altieri Breast Cancer Research 2008, 10:R97 (24 November 2008)

Notch-1-survivin functional gene signature validation as a basal breast cancer hallmark suggests novel roles for Notch and survivin antagonists as molecular therapies against this recurrence-prone disease type.