Table 1

Summary of linkage scan on chromosome 15q14-22.1

Population
Sample
Score
Phenotype
STS marker
Position (cM)
References (year)

Japanese
224 ASP in 159 families, 359 individuals
1.57 MLS
T2D
D15S994
40.25
Mori Y et al. 2002 [11]


3.91 MLS
T2D (Young-onset < 45)
D15S994
40.25



2.44 MLS
T2D (BMI < 30)
D15S994
40.25



1.50 MLS
T2D
D15S118
32.58



0.95 MLS
T2D
D15S117
51.21

Japanese
256 ASP in 164 families, 368 individuals
2.41 LOD
T2D (BMI < 22)
CYP19
45.62
Iwasaki N et al. 2003 [12]
Japanese
102 ASP in 102 families
0.25 LOD
T2D
D15S117
51.21
Nawata H et al. 2004 [13]

Mexican Americans
330 ASP in 170 families, 408 individuals
1.50 MLS
T2D
D15S119
45.62
Hanis CL et al. 1996 [14]
Pima Indians
388 ASP in 109 families, 363 individuals
1.46 LOD
insulin secretion (IVGTT 10)
D15S659
43.47
Pratley RE et al. 1998 [15]
Mexican Americans
330 ASP in 170 families and 363 nondiabetic individuals
1.27 LOD
T2D
D15S119
45.62
Cox NJ et al. 1999 [16]

interaction with D15S119 and CYP19
4.00 LOD
T2D
D15S119+ CYP19



ASP, affected sib-pairs; MLS, maximum lod score; LOD, logarithm of odds; IVGTT 10, plasma insulin response at 10 min above the basal level during the intravenous glucose tolerance test; D15S119 + CYP19, D15S119 interaction with CYP19 (NIDDM1). Each report identified nominal evidence of linkage or significant evidence of linkage with LOD > 3.91. Location is shown only for loci with positive evidence of linkage on chromosome 15q. The position is based on the Marshfield recombination genetic map .

http://research.marshfieldclinic.org/genetics/Home/index.asp

Yamaguchi et al. BMC Medical Genetics 2008 9:22   doi:10.1186/1471-2350-9-22