Table 5

Synergistic effect of AGT235 MM and AT1R CC genotypes and classical risk factors in CAD patients and controls

Classical risk factor
AGT235 MM
Whole population1
AT1R CC
Whole population1

Hypertension

CAD
Controls
OR(95%CI)

CAD
Controls
OR(95%CI)





0
0
88
293
1
0
99
334
1
0
1
18
62
0.97(0.52–1.78)NS
1
7
21
1.12(0.42–2.89)NS
1
0
153
130
3.92(2.77–5.55)***
0
182
150
4.09(2.96–5.66)***
1
1
39
25
5.19(2.88–9.41)***
1
10
5
6.75(2.06–23.27)**


SI = 1.44; SIM = 1.36; RERI = 1.30; AP = 0.25
SI = 1.79; SIM = 1.47; RERI = 2.54; AP = 0.38
Smoking








0
0
145
315
1
0
177
356
1
0
1
37
58
1.39(0.86–2.24)NS
1
5
17
0.59(0.19–1.74)NS
1
0
96
108
1.93(1.36–2.75)**
0
104
128
1.63(1.18–2.27)**
1
1
20
29
1.50(0.79–2.85)NS
1
12
9
2.68(1.03–7.05)*


SI = 0.38; SIM = 0.56; RERI = -0.82; AP = -0.55
SI = 7.64; SIM = 2.79; RERI = 1.46; AP = 0.54
Obesity








0
0
172
352
1
0
201
394
1
0
1
42
65
1.32(0.84–2.07)NS
1
13
23
1.11(0.52–2.34)NS
1
0
69
71
1.99(1.34–2.95)**
0
80
90
1.74(1.22–2.50)**
1
1
15
22
1.40(0.67–2.89)NS
1
4
3
2.61(0.49–14.83)NS


SI = 0.31; SIM = 0.53; RERI = -0.91; AP = -0.65
SI = 1.89; SIM = 1.35; RERI = 0.76; AP = 0.29
Diabetes








0
0
171
396
1
0
201
450
1
0
1
41
80
1.19(0.77–1.84)NS
1
11
26
0.95(0.43–2.05)NS
1
0
70
27
6.00(3.63–9.98)***
0
80
34
5.27(3.34–8.33)***
1
1
16
7
5.29(2.00–14.47)***
1
6
0
--


SI = 0.83; SIM = 0.74; RERI = -0.90; AP = -0.17
--
Dyslipidaemia








0
0
73
370
1
0
82
424
1
0
1
14
80
0.89(0.45–1.71)NS
1
5
26
0.99(0.32–2.83)NS
1
0
168
53
16.07(10.59–24.43)***
0
199
60
17.15(11.62–25.37)***
1
1
43
7
31.14(12.81–79.15)***
1
12
0
--


SI = 2.01; SIM = 2.18; RERI = 15.18; AP = 0.49
--

SI, Rothman's synergy index for interaction; RERI, relative excess risk due to interaction; AP, proportion of disease attributable to interaction; *P < 0.05, **P < 0.005, ***P < 0.0001; 1We performed no further subdivision when analysing these polymorphisms because several classes resulted statistically impossible to analyse due to the reduced number of individuals there included.

Freitas et al. BMC Cardiovascular Disorders 2008 8:15   doi:10.1186/1471-2261-8-15