Genetic interactors were found to cause shortened rhabdomeres with Dys and Dg. (A) Dys and Dg homozygous mutants have significantly shorter rhabdomeres in the developed adult eye and interact in this process. Measurements have been normalized to control. (B) Genetic interactions found with Dys, and with Dg normalized to Dys/+ or Dg/+ appropriately (C). Rhabdomere examples from OregonR (WT,D), Camn339/+;DysDf/+ (E) and captE636/+;DysDf/+ (F). Scale bare represents wt rhabdomere length. Statistics were determined using a one-tailed Student's t-test where ***p ≤ 0.001, **p ≤ 0.01, *p ≤ 0.05.
Marrone et al. BMC Neuroscience 2011 12:93 doi:10.1186/1471-2202-12-93