Research article
Removal of Hsf4 leads to cataract development in mice through down-regulation of γS-crystallin and Bfsp expression
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* Corresponding authors: Xiangyin Kong xykong@sibs.ac.cn - Landian Hu ldhu@srcb.ac.cn
1 Institute of Health Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences and Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, PR China
2 State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiaotong University, 197 Rui Jin Road II, Shanghai 200025, PR China
BMC Molecular Biology 2009, 10:10 doi:10.1186/1471-2199-10-10
Published: 19 February 2009Additional files
Additional file 1:
Lack of Hsf4 worsens the lens fiber defect in γS-crystallin mutation mouse rncat. Hematoxylin and eosin stain revealed an aggravated lens fiber defect in 8-week-old heterozygous and homozygous rncat mice in the absence of the Hsf4 gene. Bar, 50 um.
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