Open Access Research article

The juvenile alopecia mutation (jal) maps to mouse Chromosome 2, and is an allele of GATA binding protein 3 (Gata3)

Francisco Ramirez, Aaron M Feliciano, Elisabeth B Adkins, Kevin M Child, Legairre A Radden II, Alexis Salas, Nelson Vila-Santana, José M Horák, Samantha R Hughes, Damek V Spacek and Thomas R King*

  • * Corresponding author: Thomas R King kingt@ccsu.edu

  • † Equal contributors

Author Affiliations

Biomolecular Sciences, Central Connecticut State University, 1615 Stanley Street, New Britain, CT 06053, USA

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BMC Genetics 2013, 14:40  doi:10.1186/1471-2156-14-40

Published: 9 May 2013

Additional files

Additional file 1:

Three-month-old mutants from a (C3H/HeJ-jal/J x C57BL/6 J)F1 × C3H/HeJ-jal/J backcross display variable expressivity of the juvenile alopecia phenotype.

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Additional file 2:

Description of SNP markers referred to in the Ramirez et al. (2013) text.

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Additional file 3:

Location of SNP markers referred to in the Ramirez et al. (2013) text.

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Additional file 4:

F1 data from reciprocal crosses in mice tests the juvenile alopecia mutation (jal) for X versus autosomal linkage.

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Additional file 5:

DNA typing for the Il2ratm1Dwor Il2ra+alleles among the progeny of a complementation cross, Il2ratm1Dw/Il2ra+x jal/jal.

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Additional file 6:

The recessive jal and Il2ratm1Dwmutations complement in doubly heterozygous mice.

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Additional file 7:

Sequence analysis of Gata3 splice junctions in wild-type C3H/HeJ and mutant C3H/HeJ-jal cDNA.

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