Table 4

Rescue of AVKR pathfinding defects
TRANSGENE GENOTYPE % AVKR DEFECT N p
none wild-type 0 27
none fax-1 (gm83) 78 32
fax-1 9 kb genomic DNA fax-1 (gm83) 12 25
none fax-1 (gm83) lin-15 (n765) 97 35
FAX-1::INV FAX-1 fax-1 (gm83) lin-15 (n765) 92 53 0.55
FAX-1::FAX-1 LBD fax-1 (gm83) lin-15 (n765) 33 52 <0.001
FAX-1::CbFAX-1 LBD fax-1 (gm83) lin-15 (n765) 36 22 <0.001
FAX-1::NHR-111 LBD fax-1 (gm83) lin-15 (n765) 61 95 <0.001
FAX-1::NHR-67 LBD fax-1 (gm83) lin-15 (n765) 41 39 <0.001
FAX-1::Δ LBD fax-1 (gm83) lin-15 (n765) 62 63 <0.001

The percentage of the AVKR axon crossover defect [56] is shown for each strain. N = number of AVKR axons examined. p = Chi square P values relative to fax-1(gm83) lin-15(n756) strain without any transgene.

Weber et al.

Weber et al. BMC Evolutionary Biology 2012 12:81   doi:10.1186/1471-2148-12-81

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